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Metformin and simvastatin play a crucial role in regulating cellular metabolic pathways Previous studies have reported that Metformin can regulate the AMPK pathway [32] and cell cycle [33], while simvastatin can modulate the mTOR [34] and SREBP pathways [35]
We do not prescribe to vulnerable populations, and we do not prescribe for cosmetic optimisation as a standalone indication
reported that in breast cancer cells, Erastin significantly increased the expression of key autophagy-associated proteins, including beclin-1, ATG5, ATG12, and LC3B, while concurrently decreasing levels of the autophagy substrate p62 (50)
Another source of reducing equivalents comes from the selective transport of GSH, rather than GSSG, from the cytosol into the ER lumen
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